Genome-wide association analyses identify 95 risk loci and provide insights into the neurobiology of post-traumatic stress disorder
Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt
Originalsprog | Engelsk |
---|---|
Tidsskrift | Nature Genetics |
Vol/bind | 56 |
Udgave nummer | 5 |
Sider (fra-til) | 792-808 |
Antal sider | 17 |
ISSN | 1061-4036 |
DOI | |
Status | Udgivet - 2024 |
Bibliografisk note
Funding Information:
Major financial support for the PTSD-PGC was provided by the National Institute of Mental Health (NIMH; R01MH106595 (to K.C.K., C.M.N., K.J.R. and M.B.S.), R01MH124847 (to C.M.N.) and R01MH124851 (to A.D.B., L.K.D. and K.C.K.)), the Stanley Center for Psychiatric Research at the Broad Institute and Cohen Veterans Bioscience. Statistical analyses were carried out on the NL Genetic Cluster computer (URL) hosted by SURFsara. Genotyping of samples was supported in part through the Stanley Center for Psychiatric Genetics at the Broad Institute of MIT and Harvard. This research has been conducted using the UKB resource under application 41209. This work would not have been possible without the contributions of the investigators who comprise the PGC-PTSD working group, and especially the more than 1,307,247 research participants worldwide who shared their life experiences and biological samples with PGC-PTSD investigators. We thank A.E. Aiello, B. Bradley, A. Gautam, R. Hammamieh, M. Jett, M.J. Lyons, D. Maurer, M.R. Mavissakalian and the late C.R. Erbes and R.E. McGlinchey for their contributions to this study.
Publisher Copyright:
© This is a U.S. Government work and not under copyright protection in the US; foreign copyright protection may apply 2024.
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