Jesper Bratz Birk
Akademisk medarbejder
August Krogh Sektionen for Molekylær Fysiologi
Nørre Allé 51, 2200 København N
ORCID: 0000-0001-9775-4789
1 - 4 ud af 4Pr. side: 25
- Udgivet
Hyperphosphorylation and low protein content of muscle glycogen synthase contribute to insulin resistance in polycystic ovary syndrome: effects of pioglitazone treatment
Hojlund, K., Glintborg, D., Andersen, N. R., Birk, Jesper Bratz, Treebak, Jonas Thue, Beck-Nielsen, H. & Wojtaszewski, Jørgen F P, 2007, I: Diabetologia.Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt
- Udgivet
Absence of humoral mediated 5'AMP-activated protein kinase activation in human skeletal muscle and adipose tissue during exercise
Kristensen, Jonas Møller, Johnsen, A. B., Birk, Jesper Bratz, Nielsen, J. N., Jensen, B. R., Hellsten, Ylva, Richter, Erik A. & Wojtaszewski, Jørgen F P, 2007, I: Journal of Physiology. 585, 3, s. 897-909 13 s.Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt
- Udgivet
AS160 phosphorylation is associated with activation of alpha(2)beta(2)gamma(1)- but not alpha(2)beta(2)gamma(3)-AMPK trimeric complex in skeletal muscle during exercise in humans
Treebak, Jonas Thue, Birk, Jesper Bratz, Rose, A. J., Kiens, Bente, Richter, Erik A. & Wojtaszewski, Jørgen F P, 2007, I: American Journal of Physiology-Endocrinology and Metabolism.Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt
- Udgivet
AS160 phosphorylation is associated with activation of alpha2beta2gamma1 - but not alpha2beta2gamma3 -AMPK trimeric complex in skeletal muscle during exercise in humans
Treebak, Jonas Thue, Birk, Jesper Bratz, Rose, A. J., Kiens, Bente, Richter, Erik A. & Wojtaszewski, Jørgen F P, 2007, I: American Journal of Physiology: Endocrinology and Metabolism. 292, 3, s. E715-E722 8 s.Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt
ID: 1841140
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AMPK and TBC1D1 regulate muscle glucose uptake after, but not during, exercise and contraction
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Exercise-induced molecular mechanisms promoting glycogen supercompensation in human skeletal muscle
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Inducible deletion of skeletal muscle AMPKα 1 reveals that AMPK is required for nucleotide balance but dispensable for muscle glucose uptake and fat oxidation during exercise
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