Trends in puberty timing in humans and environmental modifiers

Forskning

Trends in puberty timing in humans and environmental modifiers

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Standard

Trends in puberty timing in humans and environmental modifiers. / Toppari, Jorma; Juul, Anders.

I: Molecular and Cellular Endocrinology, Bind 324, Nr. 1-2, 05.08.2010, s. 39-44.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Toppari, J & Juul, A 2010, 'Trends in puberty timing in humans and environmental modifiers', Molecular and Cellular Endocrinology, bind 324, nr. 1-2, s. 39-44. https://doi.org/10.1016/j.mce.2010.03.011

APA

Toppari, J., & Juul, A. (2010). Trends in puberty timing in humans and environmental modifiers. Molecular and Cellular Endocrinology, 324(1-2), 39-44. https://doi.org/10.1016/j.mce.2010.03.011

Vancouver

Toppari J, Juul A. Trends in puberty timing in humans and environmental modifiers. Molecular and Cellular Endocrinology. 2010 aug. 5;324(1-2):39-44. https://doi.org/10.1016/j.mce.2010.03.011

Author

Toppari, Jorma ; Juul, Anders. / Trends in puberty timing in humans and environmental modifiers. I: Molecular and Cellular Endocrinology. 2010 ; Bind 324, Nr. 1-2. s. 39-44.

Bibtex

@article{abd0ecc9a1e34d9fb3bd02b1d1e5384e,

title = "Trends in puberty timing in humans and environmental modifiers",

abstract = "Secular trends in timing of puberty appear to continue although under-nutrition has not been any longer a limiting factor for pubertal development. Now obesity and other environmental reasons have been suspected to cause this trend, and endocrine disrupting chemicals have become into focus as possible contributors. Epidemiological studies on endocrine disrupters are still scarce and show only weak associations between exposures and timing of puberty. Since genetic background explains 50-80% of variability in the timing of puberty, it is not surprising that the observed environmental effects are rather modest when individual exposures are assessed. Despite that, some exposures have been reported to be associated to early (e.g., polybrominated biphenyls) or delayed (e.g., lead) puberty. Here we shortly review the available data on recent trends in timing of puberty and the possible role of endocrine disrupters.",

author = "Jorma Toppari and Anders Juul",

year = "2010",

month = aug,

day = "5",

doi = "http://dx.doi.org/10.1016/j.mce.2010.03.011",

language = "English",

volume = "324",

pages = "39--44",

journal = "Molecular and Cellular Endocrinology",

issn = "0303-7207",

publisher = "Elsevier Ireland Ltd",

number = "1-2",

}

RIS

TY - JOUR

T1 - Trends in puberty timing in humans and environmental modifiers

AU - Toppari, Jorma

AU - Juul, Anders

PY - 2010/8/5

Y1 - 2010/8/5

N2 - Secular trends in timing of puberty appear to continue although under-nutrition has not been any longer a limiting factor for pubertal development. Now obesity and other environmental reasons have been suspected to cause this trend, and endocrine disrupting chemicals have become into focus as possible contributors. Epidemiological studies on endocrine disrupters are still scarce and show only weak associations between exposures and timing of puberty. Since genetic background explains 50-80% of variability in the timing of puberty, it is not surprising that the observed environmental effects are rather modest when individual exposures are assessed. Despite that, some exposures have been reported to be associated to early (e.g., polybrominated biphenyls) or delayed (e.g., lead) puberty. Here we shortly review the available data on recent trends in timing of puberty and the possible role of endocrine disrupters.

AB - Secular trends in timing of puberty appear to continue although under-nutrition has not been any longer a limiting factor for pubertal development. Now obesity and other environmental reasons have been suspected to cause this trend, and endocrine disrupting chemicals have become into focus as possible contributors. Epidemiological studies on endocrine disrupters are still scarce and show only weak associations between exposures and timing of puberty. Since genetic background explains 50-80% of variability in the timing of puberty, it is not surprising that the observed environmental effects are rather modest when individual exposures are assessed. Despite that, some exposures have been reported to be associated to early (e.g., polybrominated biphenyls) or delayed (e.g., lead) puberty. Here we shortly review the available data on recent trends in timing of puberty and the possible role of endocrine disrupters.

U2 - http://dx.doi.org/10.1016/j.mce.2010.03.011

DO - http://dx.doi.org/10.1016/j.mce.2010.03.011

M3 - Journal article

VL - 324

SP - 39

EP - 44

JO - Molecular and Cellular Endocrinology

JF - Molecular and Cellular Endocrinology

SN - 0303-7207

IS - 1-2

ER -

ID: 34047666