Standard
The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes. / Gaster, Michael; Brusgaard, Klaus; Handberg, Aa.; Højlund, K.; Wojtaszewski, Jørgen; Beck-Nielsen, H.
I:
Biochemical and Biophysical Research Communications, Bind 319, Nr. 4, 2004, s. 1235-1240.
Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt
Harvard
Gaster, M, Brusgaard, K, Handberg, A, Højlund, K
, Wojtaszewski, J & Beck-Nielsen, H 2004, '
The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes',
Biochemical and Biophysical Research Communications, bind 319, nr. 4, s. 1235-1240.
https://doi.org/10.1016/j.bbrc.2004.05.109
APA
Gaster, M., Brusgaard, K., Handberg, A., Højlund, K.
, Wojtaszewski, J., & Beck-Nielsen, H. (2004).
The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes.
Biochemical and Biophysical Research Communications,
319(4), 1235-1240.
https://doi.org/10.1016/j.bbrc.2004.05.109
Vancouver
Gaster M, Brusgaard K, Handberg A, Højlund K
, Wojtaszewski J, Beck-Nielsen H.
The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes.
Biochemical and Biophysical Research Communications. 2004;319(4):1235-1240.
https://doi.org/10.1016/j.bbrc.2004.05.109
Author
Gaster, Michael ; Brusgaard, Klaus ; Handberg, Aa. ; Højlund, K. ; Wojtaszewski, Jørgen ; Beck-Nielsen, H. / The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes. I: Biochemical and Biophysical Research Communications. 2004 ; Bind 319, Nr. 4. s. 1235-1240.
Bibtex
@article{64bee410dde611dbbee902004c4f4f50,
title = "The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes",
author = "Michael Gaster and Klaus Brusgaard and Aa. Handberg and K. H{\o}jlund and J{\o}rgen Wojtaszewski and H. Beck-Nielsen",
note = "PUF 2004 5200 069",
year = "2004",
doi = "10.1016/j.bbrc.2004.05.109",
language = "English",
volume = "319",
pages = "1235--1240",
journal = "Biochemical and Biophysical Research Communications",
issn = "0006-291X",
publisher = "Elsevier",
number = "4",
}
RIS
TY - JOUR
T1 - The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes
AU - Gaster, Michael
AU - Brusgaard, Klaus
AU - Handberg, Aa.
AU - Højlund, K.
AU - Wojtaszewski, Jørgen
AU - Beck-Nielsen, H.
N1 - PUF 2004 5200 069
PY - 2004
Y1 - 2004
U2 - 10.1016/j.bbrc.2004.05.109
DO - 10.1016/j.bbrc.2004.05.109
M3 - Journal article
C2 - 15194499
VL - 319
SP - 1235
EP - 1240
JO - Biochemical and Biophysical Research Communications
JF - Biochemical and Biophysical Research Communications
SN - 0006-291X
IS - 4
ER -