OTULIN deficiency causes auto-inflammatory syndrome
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OTULIN deficiency causes auto-inflammatory syndrome. / Fiil, Berthe Katrine; Gyrd-Hansen, Mads.
I: Cell Research, Bind 26, Nr. 11, 11.2016, s. 1176-1177.Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Rådgivning
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TY - JOUR
T1 - OTULIN deficiency causes auto-inflammatory syndrome
AU - Fiil, Berthe Katrine
AU - Gyrd-Hansen, Mads
PY - 2016/11
Y1 - 2016/11
N2 - Ubiquitin chains assembled via the N-terminal methionine (Met1 or linear ubiquitin), conjugated by the linear ubiquitin chain assembly complex (LUBAC), participate in NF-κΒ-dependent inflammatory signaling and immune responses. A recent report in Cell finds that OTULIN, a deubiquitinase that selectively cleaves Met1-linked ubiquitin chains, is essential for restraining inflammation in vivo.
AB - Ubiquitin chains assembled via the N-terminal methionine (Met1 or linear ubiquitin), conjugated by the linear ubiquitin chain assembly complex (LUBAC), participate in NF-κΒ-dependent inflammatory signaling and immune responses. A recent report in Cell finds that OTULIN, a deubiquitinase that selectively cleaves Met1-linked ubiquitin chains, is essential for restraining inflammation in vivo.
KW - Endopeptidases
KW - Immunity, Innate
KW - Lymphocytes
KW - Neurotransmitter Agents
KW - Ubiquitin
U2 - 10.1038/cr.2016.113
DO - 10.1038/cr.2016.113
M3 - Journal article
C2 - 27686184
VL - 26
SP - 1176
EP - 1177
JO - Cell Research
JF - Cell Research
SN - 1001-0602
IS - 11
ER -
ID: 280717847