Natural history of COPD

Forskning

Natural history of COPD: Focusing on change in FEV₁

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Standard

Natural history of COPD : Focusing on change in FEV₁. / Vestbo, Jørgen; Lange, Peter.

I: Respirology, Bind 21, Nr. 1, 01.2016, s. 34-43.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Vestbo, J & Lange, P 2016, 'Natural history of COPD: Focusing on change in FEV₁', Respirology, bind 21, nr. 1, s. 34-43. https://doi.org/10.1111/resp.12589

APA

Vestbo, J., & Lange, P. (2016). Natural history of COPD: Focusing on change in FEV₁. Respirology, 21(1), 34-43. https://doi.org/10.1111/resp.12589

Vancouver

Vestbo J, Lange P. Natural history of COPD: Focusing on change in FEV₁. Respirology. 2016 jan.;21(1):34-43. https://doi.org/10.1111/resp.12589

Author

Vestbo, Jørgen ; Lange, Peter. / Natural history of COPD : Focusing on change in FEV₁. I: Respirology. 2016 ; Bind 21, Nr. 1. s. 34-43.

Bibtex

@article{cd08fd4f5e7b4c8fadd35275445abd99,

title = "Natural history of COPD: Focusing on change in FEV1",

abstract = "The natural history of chronic obstructive pulmonary disease (COPD) is usually described with a focus on change in forced expiratory volume in 1 s (FEV1 ) over time as this allows for exploration of risk factors for an accelerated decline-and thus of developing COPD. From epidemiological studies we have recognized important risk factors such as smoking, exposure to biomass and occupational exposures, but we have also learnt about features such as chronic bronchitis, airway hyper-responsiveness and asthma that seem to accelerate decline in FEV1 independent of exposures. In addition we are gradually beginning to better link early life events to subsequent risk of disease in adulthood. Although more complicated, our current understanding of COPD has come a long way from being a simple image of smoking leading to poor lungs.",

author = "J{\o}rgen Vestbo and Peter Lange",

note = "{\textcopyright} 2015 Asian Pacific Society of Respirology.",

year = "2016",

month = jan,

doi = "10.1111/resp.12589",

language = "English",

volume = "21",

pages = "34--43",

journal = "Respirology",

issn = "1323-7799",

publisher = "Wiley",

number = "1",

}

RIS

TY - JOUR

T1 - Natural history of COPD

T2 - Focusing on change in FEV1

AU - Vestbo, Jørgen

AU - Lange, Peter

PY - 2016/1

Y1 - 2016/1

N2 - The natural history of chronic obstructive pulmonary disease (COPD) is usually described with a focus on change in forced expiratory volume in 1 s (FEV1 ) over time as this allows for exploration of risk factors for an accelerated decline-and thus of developing COPD. From epidemiological studies we have recognized important risk factors such as smoking, exposure to biomass and occupational exposures, but we have also learnt about features such as chronic bronchitis, airway hyper-responsiveness and asthma that seem to accelerate decline in FEV1 independent of exposures. In addition we are gradually beginning to better link early life events to subsequent risk of disease in adulthood. Although more complicated, our current understanding of COPD has come a long way from being a simple image of smoking leading to poor lungs.

AB - The natural history of chronic obstructive pulmonary disease (COPD) is usually described with a focus on change in forced expiratory volume in 1 s (FEV1 ) over time as this allows for exploration of risk factors for an accelerated decline-and thus of developing COPD. From epidemiological studies we have recognized important risk factors such as smoking, exposure to biomass and occupational exposures, but we have also learnt about features such as chronic bronchitis, airway hyper-responsiveness and asthma that seem to accelerate decline in FEV1 independent of exposures. In addition we are gradually beginning to better link early life events to subsequent risk of disease in adulthood. Although more complicated, our current understanding of COPD has come a long way from being a simple image of smoking leading to poor lungs.

U2 - 10.1111/resp.12589

DO - 10.1111/resp.12589

M3 - Journal article

C2 - 26176980

VL - 21

SP - 34

EP - 43

JO - Respirology

JF - Respirology

SN - 1323-7799

IS - 1

ER -

ID: 153787807