Kliniske symptomer og patofysiologi ved migraene

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Standard

Kliniske symptomer og patofysiologi ved migraene. / Tfelt-Hansen, P.; Ashina, M.; Olesen, J.; Tfelt-Hansen, Peer; Ashina, Messoud; Olesen, Jes.

I: Ugeskrift for læger, Bind 170, Nr. 41, 2008, s. 3231-4.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Tfelt-Hansen, P, Ashina, M, Olesen, J, Tfelt-Hansen, P, Ashina, M & Olesen, J 2008, 'Kliniske symptomer og patofysiologi ved migraene', Ugeskrift for læger, bind 170, nr. 41, s. 3231-4.

APA

Tfelt-Hansen, P., Ashina, M., Olesen, J., Tfelt-Hansen, P., Ashina, M., & Olesen, J. (2008). Kliniske symptomer og patofysiologi ved migraene. Ugeskrift for læger, 170(41), 3231-4.

Vancouver

Tfelt-Hansen P, Ashina M, Olesen J, Tfelt-Hansen P, Ashina M, Olesen J. Kliniske symptomer og patofysiologi ved migraene. Ugeskrift for læger. 2008;170(41):3231-4.

Author

Tfelt-Hansen, P. ; Ashina, M. ; Olesen, J. ; Tfelt-Hansen, Peer ; Ashina, Messoud ; Olesen, Jes. / Kliniske symptomer og patofysiologi ved migraene. I: Ugeskrift for læger. 2008 ; Bind 170, Nr. 41. s. 3231-4.

Bibtex

@article{59e3bc90064911deb05e000ea68e967b,
title = "Kliniske symptomer og patofysiologi ved migraene",
abstract = "Understanding of the pathophysiology of migraine attacks requires consideration of both symptoms and paraclinical investigations. During the aura phase, characteristic changes in regional cerebral blood flow (rCBF) can be observed. The headache is most likely a form of neurovascular pain. Nausea, photo- and phonophobia are probably elicited from the central nervous system. Positron emission tomography (PET) scans reveal specific changes in the brain stem. Allodynia, most likely caused by central sensitization, often develops during the attack. In conclusion, the migraine attack is a complex neurobiological and neurovascular process. Udgivelsesdato: 2008-Oct-6",
author = "P. Tfelt-Hansen and M. Ashina and J. Olesen and Peer Tfelt-Hansen and Messoud Ashina and Jes Olesen",
note = "Keywords: Brain; Humans; Migraine Disorders; Migraine with Aura; Positron-Emission Tomography",
year = "2008",
language = "Dansk",
volume = "170",
pages = "3231--4",
journal = "Ugeskrift for Laeger",
issn = "0041-5782",
publisher = "Almindelige Danske Laegeforening",
number = "41",

}

RIS

TY - JOUR

T1 - Kliniske symptomer og patofysiologi ved migraene

AU - Tfelt-Hansen, P.

AU - Ashina, M.

AU - Olesen, J.

AU - Tfelt-Hansen, Peer

AU - Ashina, Messoud

AU - Olesen, Jes

N1 - Keywords: Brain; Humans; Migraine Disorders; Migraine with Aura; Positron-Emission Tomography

PY - 2008

Y1 - 2008

N2 - Understanding of the pathophysiology of migraine attacks requires consideration of both symptoms and paraclinical investigations. During the aura phase, characteristic changes in regional cerebral blood flow (rCBF) can be observed. The headache is most likely a form of neurovascular pain. Nausea, photo- and phonophobia are probably elicited from the central nervous system. Positron emission tomography (PET) scans reveal specific changes in the brain stem. Allodynia, most likely caused by central sensitization, often develops during the attack. In conclusion, the migraine attack is a complex neurobiological and neurovascular process. Udgivelsesdato: 2008-Oct-6

AB - Understanding of the pathophysiology of migraine attacks requires consideration of both symptoms and paraclinical investigations. During the aura phase, characteristic changes in regional cerebral blood flow (rCBF) can be observed. The headache is most likely a form of neurovascular pain. Nausea, photo- and phonophobia are probably elicited from the central nervous system. Positron emission tomography (PET) scans reveal specific changes in the brain stem. Allodynia, most likely caused by central sensitization, often develops during the attack. In conclusion, the migraine attack is a complex neurobiological and neurovascular process. Udgivelsesdato: 2008-Oct-6

M3 - Tidsskriftartikel

VL - 170

SP - 3231

EP - 3234

JO - Ugeskrift for Laeger

JF - Ugeskrift for Laeger

SN - 0041-5782

IS - 41

ER -

ID: 10949474