Inflammatory single nucleotide polymorphisms and the risk of atrial fibrillation: a case control study

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Inflammatory single nucleotide polymorphisms and the risk of atrial fibrillation: a case control study. / Henningsen, Kristoffer M; Olesen, Morten Sig; Ravn, Lasse S; Dixen, Ulrik; Haunsoe, Stig; Bruunsgaard, Helle; Svendsen, Jesper Hastrup.

I: Inflammation Research, Bind 60, Nr. 3, 03.2011, s. 209-211.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Henningsen, KM, Olesen, MS, Ravn, LS, Dixen, U, Haunsoe, S, Bruunsgaard, H & Svendsen, JH 2011, 'Inflammatory single nucleotide polymorphisms and the risk of atrial fibrillation: a case control study', Inflammation Research, bind 60, nr. 3, s. 209-211. https://doi.org/10.1007/s00011-010-0285-2

APA

Henningsen, K. M., Olesen, M. S., Ravn, L. S., Dixen, U., Haunsoe, S., Bruunsgaard, H., & Svendsen, J. H. (2011). Inflammatory single nucleotide polymorphisms and the risk of atrial fibrillation: a case control study. Inflammation Research, 60(3), 209-211. https://doi.org/10.1007/s00011-010-0285-2

Vancouver

Henningsen KM, Olesen MS, Ravn LS, Dixen U, Haunsoe S, Bruunsgaard H o.a. Inflammatory single nucleotide polymorphisms and the risk of atrial fibrillation: a case control study. Inflammation Research. 2011 mar.;60(3):209-211. https://doi.org/10.1007/s00011-010-0285-2

Author

Henningsen, Kristoffer M ; Olesen, Morten Sig ; Ravn, Lasse S ; Dixen, Ulrik ; Haunsoe, Stig ; Bruunsgaard, Helle ; Svendsen, Jesper Hastrup. / Inflammatory single nucleotide polymorphisms and the risk of atrial fibrillation: a case control study. I: Inflammation Research. 2011 ; Bind 60, Nr. 3. s. 209-211.

Bibtex

@article{dbac5a4952cf41708805b74a2d17c02b,
title = "Inflammatory single nucleotide polymorphisms and the risk of atrial fibrillation: a case control study",
abstract = "INTRODUCTION: Systemic inflammation is associated with atrial fibrillation (AF) and inflammatory processes are involved in the pathophysiology of AF. We hypothesized that genetic polymorphisms, which determine the rate of inflammatory cytokines, are associated with increased risk of AF. MATERIALS AND METHODS: We included 158 patients with AF and 188 healthy controls. All patients were genotyped for common single nucleotide polymorphisms (SNPs) in selected inflammatory genes. RESULTS: A case-control analysis of the investigated SNPs (IL1A-889, TNF-308, IL1B-511, IL10-592, IL10-1082, IL18-137 and IL18-607) revealed no significant differences in the frequencies of genotypes between the AF patients and the healthy controls.",
author = "Henningsen, {Kristoffer M} and Olesen, {Morten Sig} and Ravn, {Lasse S} and Ulrik Dixen and Stig Haunsoe and Helle Bruunsgaard and Svendsen, {Jesper Hastrup}",
year = "2011",
month = mar,
doi = "10.1007/s00011-010-0285-2",
language = "English",
volume = "60",
pages = "209--211",
journal = "Inflammation Research",
issn = "1023-3830",
publisher = "Springer Basel AG",
number = "3",

}

RIS

TY - JOUR

T1 - Inflammatory single nucleotide polymorphisms and the risk of atrial fibrillation: a case control study

AU - Henningsen, Kristoffer M

AU - Olesen, Morten Sig

AU - Ravn, Lasse S

AU - Dixen, Ulrik

AU - Haunsoe, Stig

AU - Bruunsgaard, Helle

AU - Svendsen, Jesper Hastrup

PY - 2011/3

Y1 - 2011/3

N2 - INTRODUCTION: Systemic inflammation is associated with atrial fibrillation (AF) and inflammatory processes are involved in the pathophysiology of AF. We hypothesized that genetic polymorphisms, which determine the rate of inflammatory cytokines, are associated with increased risk of AF. MATERIALS AND METHODS: We included 158 patients with AF and 188 healthy controls. All patients were genotyped for common single nucleotide polymorphisms (SNPs) in selected inflammatory genes. RESULTS: A case-control analysis of the investigated SNPs (IL1A-889, TNF-308, IL1B-511, IL10-592, IL10-1082, IL18-137 and IL18-607) revealed no significant differences in the frequencies of genotypes between the AF patients and the healthy controls.

AB - INTRODUCTION: Systemic inflammation is associated with atrial fibrillation (AF) and inflammatory processes are involved in the pathophysiology of AF. We hypothesized that genetic polymorphisms, which determine the rate of inflammatory cytokines, are associated with increased risk of AF. MATERIALS AND METHODS: We included 158 patients with AF and 188 healthy controls. All patients were genotyped for common single nucleotide polymorphisms (SNPs) in selected inflammatory genes. RESULTS: A case-control analysis of the investigated SNPs (IL1A-889, TNF-308, IL1B-511, IL10-592, IL10-1082, IL18-137 and IL18-607) revealed no significant differences in the frequencies of genotypes between the AF patients and the healthy controls.

U2 - 10.1007/s00011-010-0285-2

DO - 10.1007/s00011-010-0285-2

M3 - Journal article

C2 - 21132345

VL - 60

SP - 209

EP - 211

JO - Inflammation Research

JF - Inflammation Research

SN - 1023-3830

IS - 3

ER -

ID: 32476198