Epigenetic Deficiencies and Replicative Stress

Epigenetic Deficiencies and Replicative Stress: Driving Cancer Cells to an Early Grave

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Standard

Epigenetic Deficiencies and Replicative Stress : Driving Cancer Cells to an Early Grave. / Shoaib, Muhammad; Sørensen, Claus Storgaard.

I: Cancer Cell, Bind 28, Nr. 5, 09.11.2015, s. 545-547.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Shoaib, M & Sørensen, CS 2015, 'Epigenetic Deficiencies and Replicative Stress: Driving Cancer Cells to an Early Grave', Cancer Cell, bind 28, nr. 5, s. 545-547. https://doi.org/10.1016/j.ccell.2015.10.009

APA

Shoaib, M., & Sørensen, C. S. (2015). Epigenetic Deficiencies and Replicative Stress: Driving Cancer Cells to an Early Grave. Cancer Cell, 28(5), 545-547. https://doi.org/10.1016/j.ccell.2015.10.009

Vancouver

Shoaib M, Sørensen CS. Epigenetic Deficiencies and Replicative Stress: Driving Cancer Cells to an Early Grave. Cancer Cell. 2015 nov. 9;28(5):545-547. https://doi.org/10.1016/j.ccell.2015.10.009

Author

Shoaib, Muhammad ; Sørensen, Claus Storgaard. / Epigenetic Deficiencies and Replicative Stress : Driving Cancer Cells to an Early Grave. I: Cancer Cell. 2015 ; Bind 28, Nr. 5. s. 545-547.

Bibtex

@article{0a8a6ec331a14fb7ac3933083a1d1bcb,

title = "Epigenetic Deficiencies and Replicative Stress: Driving Cancer Cells to an Early Grave",

abstract = "Cancer cell-specific synthetic lethal interactions entail promising therapeutic possibilities. In this issue of Cancer Cell, Pfister et al. describe a synthetic lethal interaction where cancer cells deficient in H3K36me3 owing to SETD2 loss-of-function mutation are strongly sensitized to inhibition of WEE1, a cell cycle controlling kinase.",

keywords = "Animals, Cell Cycle Proteins, Histones, Humans, Neoplasms, Nuclear Proteins, Nucleotides, Protein-Tyrosine Kinases, Comment, Journal Article",

author = "Muhammad Shoaib and S{\o}rensen, {Claus Storgaard}",

year = "2015",

month = nov,

day = "9",

doi = "10.1016/j.ccell.2015.10.009",

language = "English",

volume = "28",

pages = "545--547",

journal = "Cancer Cell",

issn = "1535-6108",

publisher = "Cell Press",

number = "5",

}

RIS

TY - JOUR

T1 - Epigenetic Deficiencies and Replicative Stress

T2 - Driving Cancer Cells to an Early Grave

AU - Shoaib, Muhammad

AU - Sørensen, Claus Storgaard

PY - 2015/11/9

Y1 - 2015/11/9

N2 - Cancer cell-specific synthetic lethal interactions entail promising therapeutic possibilities. In this issue of Cancer Cell, Pfister et al. describe a synthetic lethal interaction where cancer cells deficient in H3K36me3 owing to SETD2 loss-of-function mutation are strongly sensitized to inhibition of WEE1, a cell cycle controlling kinase.

AB - Cancer cell-specific synthetic lethal interactions entail promising therapeutic possibilities. In this issue of Cancer Cell, Pfister et al. describe a synthetic lethal interaction where cancer cells deficient in H3K36me3 owing to SETD2 loss-of-function mutation are strongly sensitized to inhibition of WEE1, a cell cycle controlling kinase.

KW - Animals

KW - Cell Cycle Proteins

KW - Histones

KW - Humans

KW - Neoplasms

KW - Nuclear Proteins

KW - Nucleotides

KW - Protein-Tyrosine Kinases

KW - Comment

KW - Journal Article

U2 - 10.1016/j.ccell.2015.10.009

DO - 10.1016/j.ccell.2015.10.009

M3 - Journal article

C2 - 26555168

VL - 28

SP - 545

EP - 547

JO - Cancer Cell

JF - Cancer Cell

SN - 1535-6108

IS - 5

ER -

ID: 165717223