Standard
Calphostin C is an inhibitor of contraction, but not insulin-stimulated glucose transport, in skeletal muscle. / Ihlemann, Jacob; Galbo, Henrik; Ploug, Thorkil.
I:
Acta Physiologica (Print Edition), Nr. 167, 1999, s. 69-75.
Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt
Harvard
Ihlemann, J, Galbo, H & Ploug, T 1999, 'Calphostin C is an inhibitor of contraction, but not insulin-stimulated glucose transport, in skeletal muscle', Acta Physiologica (Print Edition), nr. 167, s. 69-75.
APA
Ihlemann, J., Galbo, H., & Ploug, T. (1999). Calphostin C is an inhibitor of contraction, but not insulin-stimulated glucose transport, in skeletal muscle. Acta Physiologica (Print Edition), (167), 69-75.
Vancouver
Ihlemann J, Galbo H, Ploug T. Calphostin C is an inhibitor of contraction, but not insulin-stimulated glucose transport, in skeletal muscle. Acta Physiologica (Print Edition). 1999;(167):69-75.
Author
Ihlemann, Jacob ; Galbo, Henrik ; Ploug, Thorkil. / Calphostin C is an inhibitor of contraction, but not insulin-stimulated glucose transport, in skeletal muscle. I: Acta Physiologica (Print Edition). 1999 ; Nr. 167. s. 69-75.
Bibtex
@article{2c4835e074c911dbbee902004c4f4f50,
title = "Calphostin C is an inhibitor of contraction, but not insulin-stimulated glucose transport, in skeletal muscle",
abstract = "Calmodulin, 2-deoxyglucose, exercise, GLUT4, protein kinase C, signal transduction",
author = "Jacob Ihlemann and Henrik Galbo and Thorkil Ploug",
year = "1999",
language = "English",
pages = "69--75",
journal = "Acta Physiologica",
issn = "1748-1708",
publisher = "Wiley-Blackwell",
number = "167",
}
RIS
TY - JOUR
T1 - Calphostin C is an inhibitor of contraction, but not insulin-stimulated glucose transport, in skeletal muscle
AU - Ihlemann, Jacob
AU - Galbo, Henrik
AU - Ploug, Thorkil
PY - 1999
Y1 - 1999
N2 - Calmodulin, 2-deoxyglucose, exercise, GLUT4, protein kinase C, signal transduction
AB - Calmodulin, 2-deoxyglucose, exercise, GLUT4, protein kinase C, signal transduction
M3 - Journal article
SP - 69
EP - 75
JO - Acta Physiologica
JF - Acta Physiologica
SN - 1748-1708
IS - 167
ER -
