The HETE Is on FFAR1 and Pancreatic Islet Cells

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Standard

The HETE Is on FFAR1 and Pancreatic Islet Cells. / Trauelsen, Mette; Lückmann, Michael; Frimurer, Thomas M; Schwartz, Thue W.

I: Cell Metabolism, Bind 27, Nr. 2, 2018, s. 273-275.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Trauelsen, M, Lückmann, M, Frimurer, TM & Schwartz, TW 2018, 'The HETE Is on FFAR1 and Pancreatic Islet Cells', Cell Metabolism, bind 27, nr. 2, s. 273-275. https://doi.org/10.1016/j.cmet.2018.01.006

APA

Trauelsen, M., Lückmann, M., Frimurer, T. M., & Schwartz, T. W. (2018). The HETE Is on FFAR1 and Pancreatic Islet Cells. Cell Metabolism, 27(2), 273-275. https://doi.org/10.1016/j.cmet.2018.01.006

Vancouver

Trauelsen M, Lückmann M, Frimurer TM, Schwartz TW. The HETE Is on FFAR1 and Pancreatic Islet Cells. Cell Metabolism. 2018;27(2):273-275. https://doi.org/10.1016/j.cmet.2018.01.006

Author

Trauelsen, Mette ; Lückmann, Michael ; Frimurer, Thomas M ; Schwartz, Thue W. / The HETE Is on FFAR1 and Pancreatic Islet Cells. I: Cell Metabolism. 2018 ; Bind 27, Nr. 2. s. 273-275.

Bibtex

@article{5d3b3dfccfe040e59d87564076dde770,

title = "The HETE Is on FFAR1 and Pancreatic Islet Cells",

abstract = "It is known but generally unappreciated that the fatty acid receptor FFAR1 (GPR40) is responsible for a major part of glucose-induced insulin secretion. This puzzling fact is now explained by Tunaru et al. (2018), who demonstrate that glucose-induced 20-hydroxyeicosatetraenoic acid (20-HETE) amplifies insulin secretion through autocrine activation of FFAR1.",

keywords = "Journal Article",

author = "Mette Trauelsen and Michael L{\"u}ckmann and Frimurer, {Thomas M} and Schwartz, {Thue W}",

year = "2018",

doi = "10.1016/j.cmet.2018.01.006",

language = "English",

volume = "27",

pages = "273--275",

journal = "Cell Metabolism",

issn = "1550-4131",

publisher = "Cell Press",

number = "2",

}

RIS

TY - JOUR

T1 - The HETE Is on FFAR1 and Pancreatic Islet Cells

AU - Trauelsen, Mette

AU - Lückmann, Michael

AU - Frimurer, Thomas M

AU - Schwartz, Thue W

PY - 2018

Y1 - 2018

N2 - It is known but generally unappreciated that the fatty acid receptor FFAR1 (GPR40) is responsible for a major part of glucose-induced insulin secretion. This puzzling fact is now explained by Tunaru et al. (2018), who demonstrate that glucose-induced 20-hydroxyeicosatetraenoic acid (20-HETE) amplifies insulin secretion through autocrine activation of FFAR1.

AB - It is known but generally unappreciated that the fatty acid receptor FFAR1 (GPR40) is responsible for a major part of glucose-induced insulin secretion. This puzzling fact is now explained by Tunaru et al. (2018), who demonstrate that glucose-induced 20-hydroxyeicosatetraenoic acid (20-HETE) amplifies insulin secretion through autocrine activation of FFAR1.

KW - Journal Article

U2 - 10.1016/j.cmet.2018.01.006

DO - 10.1016/j.cmet.2018.01.006

M3 - Journal article

C2 - 29414682

VL - 27

SP - 273

EP - 275

JO - Cell Metabolism

JF - Cell Metabolism

SN - 1550-4131

IS - 2

ER -

ID: 189764824