Reply to: Hultström et al., Genetic determinants of mannose-binding lectin activity predispose to thromboembolic complications in critical COVID-19. Mannose-binding lectin genetics in COVID-19

Publikation: Bidrag til tidsskriftLetterfagfællebedømt

  • Rosanna Asselta
  • Elvezia Maria Paraboschi
  • Matteo Stravalaci
  • Pietro Invernizzi
  • Paolo Bonfanti
  • Andrea Biondi
  • Isabel Pagani
  • Mattia Pedotti
  • Andrea Doni
  • Francesco Scavello
  • Sarah N. Mapelli
  • Marina Sironi
  • Chiara Perucchini
  • Luca Varani
  • Milos Matkovic
  • Andrea Cavalli
  • Daniela Cesana
  • Pierangela Gallina
  • Nicoletta Pedemonte
  • Valeria Capurro
  • Nicola Clementi
  • Nicasio Mancini
  • Rino Rappuoli
  • Stefano Duga
  • Barbara Bottazzi
  • Mariagrazia Uguccioni
  • Elisa Vicenzi
  • Alberto Mantovani
  • Cecilia Garlanda
OriginalsprogEngelsk
TidsskriftNature Immunology
Vol/bind23
Udgave nummer6
Sider (fra-til)865-867
Antal sider3
ISSN1529-2908
DOI
StatusUdgivet - 2022

Bibliografisk note

Funding Information:
This work was conducted in the framework of, and made possible by, the collective effort of the Humanitas COVID-19 Task Force, the Humanitas Gavazzeni COVID-19 Task Force and the COVID-19 Storm trial. This paper is dedicated to S. Duga, who passed on 10 November 2021 and had made a key contribution to the genetic section of this report. This work was supported by a philanthropic donation by Dolce & Gabbana fashion house (to A.M. and C.G.), by the Italian Ministry of Health for COVID-19 (grant no. COVID-2020-12371640 to A.M. and C.G.), by the Italian Ministry of University and Research (to P.I.), by the Department of Excellence project PREMIA (PREcision MedIcine Approach: bringing biomarker research to the clinic, to P.I.) and by Fondazione Cariplo (Biobanking of COVID-19 patient samples to support national and international research, to A.B.). We also thank the Banca Intesa San Paolo for their generous contribution (to R.A.) and AMAF Monza ONLUS and AIRCS for unrestricted research funding. L.V. received funding from the European Union’s Horizon 2020 research and innovation program under grant agreement no. 101003650 and from the Swiss National Science Foundation (grant no. 31003A_182270).

Funding Information:
This work was conducted in the framework of, and made possible by, the collective effort of the Humanitas COVID-19 Task Force, the Humanitas Gavazzeni COVID-19 Task Force and the COVID-19 Storm trial. This paper is dedicated to S. Duga, who passed on 10 November 2021 and had made a key contribution to the genetic section of this report. This work was supported by a philanthropic donation by Dolce & Gabbana fashion house (to A.M. and C.G.), by the Italian Ministry of Health for COVID-19 (grant no. COVID-2020-12371640 to A.M. and C.G.), by the Italian Ministry of University and Research (to P.I.), by the Department of Excellence project PREMIA (PREcision MedIcine Approach: bringing biomarker research to the clinic, to P.I.) and by Fondazione Cariplo (Biobanking of COVID-19 patient samples to support national and international research, to A.B.). We also thank the Banca Intesa San Paolo for their generous contribution (to R.A.) and AMAF Monza ONLUS and AIRCS for unrestricted research funding. L.V. received funding from the European Union’s Horizon 2020 research and innovation program under grant agreement no. 101003650 and from the Swiss National Science Foundation (grant no. 31003A_182270).

ID: 325465337