A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation

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Standard

A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation. / Olesen, Morten Steen Salling; Refsgaard, Lena; Holst, Anders Gaarsdal; Larsen, Anders Peter; Grubb, Søren; Haunsø, Stig; Svendsen, Jesper Hastrup; Olesen, Søren-Peter; Schmitt, Nicole; Callø, Kirstine.

I: Cardiovascular Research, Bind 98, Nr. 3, 2013, s. 488-495.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Olesen, MSS, Refsgaard, L, Holst, AG, Larsen, AP, Grubb, S, Haunsø, S, Svendsen, JH, Olesen, S-P, Schmitt, N & Callø, K 2013, 'A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation', Cardiovascular Research, bind 98, nr. 3, s. 488-495. https://doi.org/10.1093/cvr/cvt028

APA

Olesen, M. S. S., Refsgaard, L., Holst, A. G., Larsen, A. P., Grubb, S., Haunsø, S., Svendsen, J. H., Olesen, S-P., Schmitt, N., & Callø, K. (2013). A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation. Cardiovascular Research, 98(3), 488-495. https://doi.org/10.1093/cvr/cvt028

Vancouver

Olesen MSS, Refsgaard L, Holst AG, Larsen AP, Grubb S, Haunsø S o.a. A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation. Cardiovascular Research. 2013;98(3):488-495. https://doi.org/10.1093/cvr/cvt028

Author

Olesen, Morten Steen Salling ; Refsgaard, Lena ; Holst, Anders Gaarsdal ; Larsen, Anders Peter ; Grubb, Søren ; Haunsø, Stig ; Svendsen, Jesper Hastrup ; Olesen, Søren-Peter ; Schmitt, Nicole ; Callø, Kirstine. / A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation. I: Cardiovascular Research. 2013 ; Bind 98, Nr. 3. s. 488-495.

Bibtex

@article{c347319981744d668289c1db6d4df326,

title = "A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation",

abstract = "AimsAtrial fibrillation (AF) is the most common cardiac arrhythmia, and early-onset lone AF has been linked to mutations in genes encoding ion channels. Mutations in the pore forming subunit K4.3 leading to an increase in the transient outward potassium current (I) have previously been associated with the Brugada Syndrome. Here we aim to determine if mutations in K4.3 or in the auxiliary subunit K Channel-Interacting Protein (KChIP) 2 are associated with early-onset lone AF.Methods and resultsTwo hundred and nine unrelated early-onset lone AF patients (",

author = "Olesen, {Morten Steen Salling} and Lena Refsgaard and Holst, {Anders Gaarsdal} and Larsen, {Anders Peter} and S{\o}ren Grubb and Stig Hauns{\o} and Svendsen, {Jesper Hastrup} and S{\o}ren-Peter Olesen and Nicole Schmitt and Kirstine Call{\o}",

year = "2013",

doi = "10.1093/cvr/cvt028",

language = "English",

volume = "98",

pages = "488--495",

journal = "Cardiovascular Research",

issn = "0008-6363",

publisher = "Oxford University Press",

number = "3",

}

RIS

TY - JOUR

T1 - A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation

AU - Olesen, Morten Steen Salling

AU - Refsgaard, Lena

AU - Holst, Anders Gaarsdal

AU - Larsen, Anders Peter

AU - Grubb, Søren

AU - Haunsø, Stig

AU - Svendsen, Jesper Hastrup

AU - Olesen, Søren-Peter

AU - Schmitt, Nicole

AU - Callø, Kirstine

PY - 2013

Y1 - 2013

N2 - AimsAtrial fibrillation (AF) is the most common cardiac arrhythmia, and early-onset lone AF has been linked to mutations in genes encoding ion channels. Mutations in the pore forming subunit K4.3 leading to an increase in the transient outward potassium current (I) have previously been associated with the Brugada Syndrome. Here we aim to determine if mutations in K4.3 or in the auxiliary subunit K Channel-Interacting Protein (KChIP) 2 are associated with early-onset lone AF.Methods and resultsTwo hundred and nine unrelated early-onset lone AF patients (

AB - AimsAtrial fibrillation (AF) is the most common cardiac arrhythmia, and early-onset lone AF has been linked to mutations in genes encoding ion channels. Mutations in the pore forming subunit K4.3 leading to an increase in the transient outward potassium current (I) have previously been associated with the Brugada Syndrome. Here we aim to determine if mutations in K4.3 or in the auxiliary subunit K Channel-Interacting Protein (KChIP) 2 are associated with early-onset lone AF.Methods and resultsTwo hundred and nine unrelated early-onset lone AF patients (

U2 - 10.1093/cvr/cvt028

DO - 10.1093/cvr/cvt028

M3 - Journal article

C2 - 23400760

AN - SCOPUS:84877958196

VL - 98

SP - 488

EP - 495

JO - Cardiovascular Research

JF - Cardiovascular Research

SN - 0008-6363

IS - 3

ER -

ID: 88093167